Visiting Scholar - OBGYN

The Ohio State University
Onsite

About The Position

The Visiting Scholar will conduct independent research under the guidance of a scientific mentor in the areas of ovarian and endometrial cancer biology, with a primary focus on extracellular vesicle (EV)-mediated mechanisms of tumorigenesis. The research aims to define how EV-associated proteins contribute to cancer initiation and progression and to identify novel biomarkers and therapeutic targets. A major focus of the laboratory is obesity-associated endometrial cancer. As obesity rates continue to rise, understanding the molecular mechanisms linking obesity to endometrial carcinogenesis has become increasingly important. Current projects investigate the role of Rab27a-mediated EV secretion and PIAS3 signaling in the initiation and progression of endometrial cancer, with the goal of developing early detection biomarkers and targeted therapeutic strategies.

Requirements

  • PhD in Biomedical Sciences, OB/GYN, Cancer Biology, or a related field.
  • One to two years of research experience in cancer biology or a closely related field.
  • Demonstrated ability to conduct productive, independent research, as evidenced by peer-reviewed publications, conference presentations, and contributions to innovative experimental approaches or translational research programs.

Nice To Haves

  • Prior experience in gynecologic oncology and molecular biology is preferred.
  • Cell and tissue culture
  • Exosome/EV isolation and characterization
  • Molecular and biochemical techniques (RT-qPCR, Western blotting, ELISA, IHC, gene transfection)
  • In vivo mouse models, particularly obesity-associated endometrial cancer models
  • Transcriptional and post-transcriptional regulatory mechanisms
  • Expertise in EV biology, EV-mediated signaling, and early-stage ovarian or endometrial cancer development.

Responsibilities

  • Conduct independent research under the guidance of a scientific mentor in the areas of ovarian and endometrial cancer biology.
  • Focus on extracellular vesicle (EV)-mediated mechanisms of tumorigenesis.
  • Define how EV-associated proteins contribute to cancer initiation and progression.
  • Identify novel biomarkers and therapeutic targets.
  • Investigate the role of Rab27a-mediated EV secretion and PIAS3 signaling in the initiation and progression of endometrial cancer.

Benefits

  • The university is an equal opportunity employer, including veterans and disability.

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What This Job Offers

Job Type

Full-time

Career Level

Senior

Education Level

Ph.D. or professional degree

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